References
A supraventricular tachycardia with a possible accessory pathway in a young Labrador Retriever
Abstract
A supraventricular tachycardia was diagnosed in a 3-year-old male Labrador Retriever. Based on patient signalment and characteristic electrocardiographic findings, an accessory pathway bypassing the normal atrioventricular nodal pathway causing an atrioventricular reciprocating tachycardia was a differential diagnosis. However, an alternative diagnosis of a focal atrial tachycardia was another possibility. The dog was successfully treated short-term with intravenous diltiazem, and long-term with modified release oral diltiazem. This report shows the utility of the electrocardiogram in the diagnosis of a supraventricular tachycardia, as well as the medical management for this emergency case.
Supraventricular tachycardias (SVTs) are fast heart rhythms originating from the sinus node, atrial tissue or atrioventricular junctional tissue. They include physiological sinus tachycardia but are often taken to refer to pathological SVTs. They can cause clinical signs such as weakness, and if sustained, can lead to tachycardia-induced cardiomyopathy and congestive heart failure (Shinbane et al, 1997). One particular type of SVT is caused by the presence of an accessory pathway of conducting tissue between the atria and ventricles, bypassing the atrioventricular node, allowing a rapid re-entrant conducting circuit to occur. This case report describes a case of a pathological SVT in a young Labrador Retriever, with differential diagnoses of an accessory pathway-mediated tachycardia or a focal atrial tachycardia.
A 3-year-old male Labrador Retriever was referred following the detection of a sustained tachycardia while undergoing investigations following a two-day history of lethargy and vomiting. Physical examination revealed a laterally recumbent, normothermic (38.3°C) dog in good body condition (37 kg). The dog had congested mucous membranes and an elevated respiratory rate (48 breaths per minute) with no adventitious lung sounds. The dog's heart rate was approximately 300 beats per minute (bpm), with a regular rhythm and no audible murmur. The femoral pulses were very hypokinetic, making possible deficits difficult to appreciate.
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